Fogorvosi szemle, 2013 (106. évfolyam, 1-4. szám)

2013-03-01 / 1. szám

26 FOGORVOSI SZEMLE 106. évf. 1. sz. 2013. hető meg; jelentős leány dominanciát találtunk a vizs­gálat során. Ez a 25%-os eltérés megfelel a nemzetközi tendenciáknak, de mégsem éri el a külföldön általáno­san tapasztalható 50%-os eltérést a fiú- és leánygyer­mekek között [13]. Moláris fogak aplasiájáról hazai vi­szonylatban Tarján számolt be [18], Megállapítható, hogy ez az aplasia irodalmi ritkaság. Összességében elmondható, hogy a vizsgálat ered­ményei néhány tekintetben változást hoznak az eddig hazánkban közölt adatokhoz képest, és az eltérések nagy része megfelel a nemzetközi szakirodalomnak. Irodalom 1. Ahmad W, Brancolini V, ul Faiyaz MF, Lam H, ul Haque S, Haider M és mtsai: A locus for autosomal recessive hypodontia with associated dental anomalies maps to chromosome 16q12.1. Am J Hum Genet 1998; 62: 987-991. 2. Alvesalo L, Portin P: The inheritance pattern of missing, peg­shaped, and strongly mesio-distally reduced upper lateral incisors. Acta Odontot Scand 1969; 27: 563-575. 3. Andlaw RS, Rock WP: A manual of peadiatric Dentistry. Churchill Livingstone 4th ed. London 1996; 155-157. 4. Arte S, Nieminen P, Apajalahti S, Haavikko K, Thesleff I, Pirinen S: Characteristics of incisor-premolar hypodontia in families. J Dent Res 2001; 80: 1445-1450. 5. Brook AH: A unifying aetiological explanation for anomalies of human tooth number and size. Arch Oral Biol 1984; 29: 373-378. 6. de Beer GR: Embryos and ancestors. Clarendon Press, Oxford 1951; 58-59. 7. Erpenstein H, Pfeiffer RA: Sex-linked-dominant hereditary re­duction in number of teeth. Humangenetik 1967; 4: 280-293. 8. Gábris K, Fábián G, Kaán M, Rózsa N, Tarján I: Prevalence of hy­podontia and hyperdontia in paedodontic and orthodontic patients in Budapest. Comm Dent Health 2006; 23: 80-82. 9. Goldenberg M, Das P, Messersmith M, Stockton DW, Patel PI, D’Souza RN: Clinical, radiographic, and genetic evaluation of a nov­el form of autosomal-dominant oligodontia. J Dent Res 2000; 79: 1469-1475. 10. Jorgenson RJ: Clinician’s view of hypodontia. J Am Dent Assoc 1980; 101:283-286. 11. Matalova E, Fleischmannova J, Sharpe PT, Tucker AS: Tooth agenesis: from molecular genetics to molecular dentistry. J Dent Res 2008; 87: 617-623. 12. Pirinen S, Kentala A, Nieminen P, Varilo T, Thesleff I, Arte S: Re­­cessively inherited lower incisor hypodontia. J Med Genet 2001; 38: 551-556. 13. Polder BJ, Van't Hof MA, Van der Linden F, Kuijpers-Jagtman AM: A meta-analysis of the prevalence of dental agenesis of permanent teeth. Community Dent Oral Epidemiol2004; Vol 32: 217-226. 14. Rolling S: Hypodontia of permanent teeth in Danish schoolchild­ren. Scan J Dent Res 1980; 88:365-369. 15. Rose JS: A survey of congenitally missing teeth, excluding third molars in 6000 orthodontic patients. Dent Pract Dent Rec 1966; 17:107-114. 16. Schalk-van der Weide Y, Beemer FA, Faber JA, Bosman F: Symp­tomatology of patients with oligodontia. J Oral Rehabil 1994; 21: 247-261. 17. Szepesi M, Nemes J, Kovalecz G, Alberth M: A csírahiányok előfordulási gyakoriságának vizsgálata a DEOEC FOK gyermek­fogászatán 1999 és 2003 között. Fogorv Szle 2006; 99: 115-119. 18. Tarján I: Az alsó első maradó moláris szimmetrikus izolált aplasiá­­ja. Fogorv Szle 1975; 68: 53-53. 19. Vastardis H, Karimbux N, Guthua SW, Seidman JG, Seidman CE: A human MSX1 homeodomain missense mutation causes selective tooth agenesis. Nat Genet 1996; 13: 417-421. 20. Yonezu T, Hayashi Y, Sasaki J, Machida Y: Prevalence of congeni­tal dental anomalies of the deciduous dentition in Japanese children. Bull Tokyo Dent Coll 1997; 38:27-32. Dr. Képes D, Dr. Gábris K: Prevalence of hypodontia at the Department of Pedodontics and Orthodontics of Semmelweis University Examination of hypodontia as one of the most frequent developmental disorder receives high importance in the national as well as in the international literature. As the disorder can affect the dental and mental development of the children therefore early diagnosis and treatment are necessary. The authors analyzed the OP radiograms of patients aged 6 to 18 who were registered between 2006 and 2011 at the Department of Pedodontics and Orthodontics of Semmelweis University, excluding children suffering from general diseases or other syndromes. The aim of the examination was to determine the occurance of missing teeth germ cases in the registered database of the Department, diagnose the prevalence of aplasia and compare the results with the national and international literature. Out of the 900 patients (372 boys and 528 girls) included in the examination missing germs were diagnosed in 94 cases (10,44%). In most cases the germ of the lower second premolars was missing (34,64%), then came the upper lateral incisors (27,38%) and the aplasia of the upper second premolars (21,23%). There was no relevant difference between the two sides. The number of affected girls was higher by 25% than the number of boys. Most of the results are close to the result in international literature. Key words: Hypodontia, aplasia, prevalence

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