Fogorvosi szemle, 2003 (96. évfolyam, 1-6. szám)
2003-08-01 / 4. szám
148 FOGORVOSI SZEMLE ■ 96. évf. 4. sz. 2003. 8. Becks H: General aspects of pyorrhoe research. Pac Dent Gaz 1929;37:259-282. 9. Black ÓV: Diseases of the peridental membrane having their beginning at the margin of the gum. In: Litch: American System of Dentistry Vol. 1. Philadelphia: Lea Brothers, 1886; 953-979. 10. Brockner RJ, Rickles NH, Porter DR: Hypophosphatasia with premature shedding of teeth and aplasia of cementum. Oral Surg Oral Med Oral Pathol 1962; 15:1351-1369. 11. Davis CG: Gum and alveolar diseases. Dental Cosmos 1879; 21: 192-201. 12. Denninson DK, Van Dyke TE: The acute inflammatory response and the role of phagocytic cells in periodontal health and disease. Periodontol20001997; 14: 54-78. 13. Glickman I: The classification of periodontal disease. In: Clinical Periodontology. 1st edn. Philadephia: W.B. Saunders Co. 1953: 481-500. 14. Goldman HM, Cohen DW: Characteristics of periodontal disease. In: Periodontal Therapy. 4th edn. St Louis C.V. Mosby Co. 1968: 75-109. 15. Gottlieb B: The formation of the pocket: Diffuse atrophy of alveolar bone. J Am Dent Assoc 1928; 15: 462-476. 16. Gottlieb B: The new concept of periodontoclasia. J Periodontol 1946; 17: 7-23. 17. Ishikawa I, Nakashima K, Kaseki T, NagasawaT, Watanabe H, Arakawa S, Nitta H, Nishihara T: Induction of the immune response in periodontopathic bacteria and its role in the pathogenesis of periodontitis. Periodontol20001997; 14:79-111. 18. JeffcoatMK, Reddy MS: Progression of probing attachment loss in adult periodontitis. J Periodontol 1991; 62:185-189. 19. Kornamn KS, Page RC, Tonetti MS: The host response to the mic robial challenge in periodontitis: assembling the players. Periodontol 20001997; 14:33-53. 20. Kornman KS: Refractory periodontitis: critical questions in clinica management. J Clin Periodontol 1996; 23: 293-298. 21. Löe H, Theilade E, Jensen SB: Experimental gingivitis in man, J Periodontol-\965\ 36:155-161. 22. Meyle J: Leukocyte adhesion deficiency and prepubertal periodontitis. Periodontol20001994; 6: 26-36. 23. Miller WD: Original Investigations Concerning Pyorrhea alveolaris. In: The Micro-Organisms of the Human Mouth. Philadelphia: The S.S. White Dental Mfg. Co. 1890: 328-334. 24. Orbán B, Weinmann JP: Diffuse atrophy of alveolar bone (periodontosis) J Periodontol 1942; 13: 31-45. 25. Orbán B: Classification and nomenclature of periodontal disease (Based on pathology, etiology and clinical picture). J Periodonto, 1942; 13:88-91. 26. Page RC, Schroeder HE: Periodontitis in man andotheranimals. A Comparative Review. Basel: S. Karger 1982. 27. Page RC, Baab DA: A new look at the etiology and pathogenesis of early onset periodontitis. Cementopathia revisited. J Periodontol 1985;56:748-751. 28. Papapanou PN: Periodontal disease. Epidemiol Ann Periodontol 1996; 1:1-36. 29. Plagmann HC, Kocher T, Kuhrau N, Celiebe A: Periodontal manifestation of hypophosphatasia. A family case report. J Clin Periodontol 1994;21:710-716. 30. Ranney RR: Position report and review of the literature. Pathogenesis of periodontal disease. In. International Conference on Research in the Biology of Periodontal Disease. Chicago: AAP 1977; 223-300. 31. Ranney RR: Classification of periodontal disease. Periodontol 20001993; 2:13-25. 32. Van der Welden U: Diagnosis of periodontitis (Letter to the editor). J Clin Periodontol 2000; 27: 960-961. Gera I: Classification of Periodontal Diseases During the past centuries the basic understanding of periodontal disease and the concepts of etiopathogenesis have evolved and substantially changed. The changes in paradigm has always been followed by the conceptual changes in the classification of periodontal disease. The earliest entirely descriptive classifications were based on the clinical signs and symptoms of periodontal disease. These were followed by a classification system based on the classical pathology paradigm established by Orbán and Gottlieb. This was replaced by the next classification reflecting the infection/host response paradigm during the late ’60s started with the pioneering works by Loe and coworkers. It is because many data indicated that the host response (determined by genetics, immunology and behavioral factors) was at least as important in the clinical manifestation of periodontal destruction than the bacterial biofilm this classification had been also updated. The AAP published its latest official classification system in 1999. Nevertheless this new system is far from perfect and should evolve as soon as enough data are collected by periodontal research to answer many controversy still existing to day. Key words: periodontitis, classification, etiopathogenesis
